Dear colleagues,
 
The question is very pertinent, and in a way may form the basis for discussions and as part of the solution.
 
My thinking is that much of what we know is partially due to improved diagnostics and reporting. PPR was for the first time suspected around 1999/2000 (I stand to be corrected) in Tanzania, but all samples screened were  negative. However, when some of these samples were re-tested in 2010, antibodies against PPRV were detected in at least 23% of the tested samples (we have observed similar outcomes with diseases such as RVF). On the other hand, there has been increased improvement in reporting methodologies, the application of local knowledge in animal health reporting/surveillance has in a way opened background doors to information which otherwise would not have been captured by routing surveillance procedures.  
 
Fredrick
 
More comments from participants please. Moderator
 
 
To unsubscribe from the FAO-AnimalHealth-L list, click the following link:
https://listserv.fao.org/cgi-bin/wa?SUBED1=FAO-AnimalHealth-L&A=1




To unsubscribe from the FAO-AnimalHealth-L list, click the following link:
https://listserv.fao.org/cgi-bin/wa?SUBED1=FAO-AnimalHealth-L&A=1